The old, the feeble, the paralytic, and paretic need special care.
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The sleeplessness of melancholia and of paretic dementia may be thus relieved.
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Occasionally alcoholic insanity takes on a paretic form, or it may be epileptic.
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During quiet breathing, equivalent tidal volumes were achieved by the paretic and healthy hemithorax.
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There may also be ataxic symptoms, paretic weakness of bowel and bladder, trembling and spasms.
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In this case the paretic segment forms the outer or receiving layer of the intussusception.
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The paretic has defects of memory, but he is, as a rule, quite unconscious of them.
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There seemed to be a great dryness of her throat and the muscles of her throat were paretic.
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These interventions are shown to enhance motor function in paretic limbs and stimulate cortical reorganization in patients with brain damage.
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No significant differences were found between the VT of the paretic and the healthy sides in patients during quiet breathing.
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To insist upon keeping a paretic all his lifetime in such an institution is highly irrational, to say the least.
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She nursed an old father in it, a bedridden mother, a paretic brother, when she should have been having children.
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The treated muscle weakens over 48–72 hours and remains paretic (partially paralyzed) for 2–4 months, at which time muscle length changes and motor fusion can stabilize the re-alignment.
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Reynolds prefers a method referred to as "paretic infusion", which involves firing a chemical projectile into the forehead of the zombie. This causes an endothermic reaction, freezing the zombie's brain and temporarily immobilizing it. When the paretic infusion method is tested on the zombie in the lab, it is only successful for a few moments, wearing off much faster than expected; the zombie breaks free and attacks a scientist, biting his fingers off before bashing his head against a wall, killing him. Infected by the zombie's bite, the scientist re-animates and attacks another technician.
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Movement abnormalities may occur among individuals with and without brain injuries due to abnormal remodeling in central nervous system. Learned non-use is an example commonly seen among patients with brain damage, such as stroke. Patients with stroke learned to suppress paretic limb movement after unsuccessful experience in paretic hand use; this may cause decreased neuronal activation at adjacent areas of the infarcted motor cortex. There are many types of therapies that are designed to overcome maladaptive plasticity in clinic and research, such as constraint-induced movement therapy (CIMT), body weight support treadmill training (BWSTT) and virtual reality therapy.
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The Man With the Miraculous Hands: The Fantastic Story of Felix Kersten, Himmler's Private Doctor, Burford Books: Springfield, NJ, 2004. ; see also Hitler the Paretic (Syphilitic) The physician Frederick Redlich however reported that there is no evidence that suggests that Hitler had syphilis.
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The symptoms of vestibulocerebellar syndrome vary among patients but are typically a unique combination of ocular abnormalities including nystagmus, poor or absent smooth pursuit (ability of the eyes to follow a moving object), strabismus (misalignment of the eyes), diplopia (double vision), oscillopsia (the sensation that stationary objects in the visual field are oscillating) and abnormal vestibulo-ocular reflex (reflex eye adjustment to stabilize gaze during head movement). Gaze-paretic nystagmus, one of the most common symptoms among patients results in poor gaze-holding due to neuron integrator dysfunction. Rebound nystagmus is also frequently found in conjunction with gaze-paretic nystagmus and is characteristic of cerebellar malfunction. These abnormal eye movements are often the earliest indicators of the disorder and may appear during childhood.
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With a diagnosis of pusher behaviour, three important variables should be seen, the most obvious of which is spontaneous body posture of a longitudinal tilt of the torso toward the paretic side of the body occurring on a regular basis and not only on occasion. The use of the nonparetic extremities to create the pathological lateral tilt of the body axis is another sign to be noted when diagnosing for pusher behaviour. This includes abduction and extension of the extremities of the non-affected side, to help in the push toward the affected (paretic) side. The third variable that is seen is that attempts of the therapist to correct the pusher posture by aiming to realign them to upright posture are resisted by the patient.
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The Parks–Bielschowsky three-step test, also known as Park's three-step test or Bielschowsky head tilt test, citing von Noorden GK: Binocular vision and ocular motility: Theory and management of strabismus. London, CV Mosby, 2002. is a method used to isolate the paretic extraocular muscle, particularly superior oblique muscle and trochlear nerve (IVth cranial nerve), in acquired vertical double vision. It was originally described by Marshall M. Parks.
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Liepert, J., Miltner, W. H. R., Bauder, H., Sommer, M., Dettmers, C., Taub, E., & Weiller, C. (1998). Motor cortex plasticity during constraint- induced movement therapy in stroke patients. Neuroscience Letters, 250, 5–8. CIMT uses constrained movement of the less-affected limb and intensive training of the paretic arm to counter-condition the nonuse of the more- affected arm learned in the acute and early sub-acute periods.
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He was responsible for the creation of the first laboratory of psychotherapy and psychoanalysis at the school of medicine at the University of Paris. His name is lent to the eponymous "Claude syndrome", which is a midbrain syndrome characterized by oculomotor palsy on the side of the lesion and ataxia on the opposite side. Also "Claude's hyperkinesis sign" is named after him -- a medical sign used to describe reflex movements of paretic muscles elicited by painful stimuli.
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Species of Alaria have complex indirect life cycles. There are two intermediate hosts involved, but there can also be paretic hosts, such as snakes, mice, birds, and humans. The eggs, which are oval, operculated, and light brown, are released from adult Alaria and excreted from the final host's faeces and hatched into miracidium that infect the snail host, in which the eggs then give rise to the asexual stage known as sporocysts. The sporocysts then produce cercariae.
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The greatest gains are seen among persons with stroke who exhibit some wrist and finger extension in the affected limb. Transcranial magnetic stimulation and brain imaging studies have demonstrated that the brain undergoes plastic changes in function and structure in patients that perform constraint induced movement therapy. These changes accompany the gains in motor function of the paretic upper limb. However, there is no established causal link between observed changes in brain function/structure and the motor gains due to constraint induced movement therapy.
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The use of the nonparetic extremities to create the pathological lateral tilt of the body axis is another sign to be noted when diagnosing for pusher behaviour. This includes abduction and extension of the extremities of the non-affected side, to help in the push toward the affected (paretic) side. The third variable that is seen is that attempts of the therapist to correct the pusher posture by aiming to realign them to upright posture are resisted by the patient. In patients with acute stroke and hemiparesis, the disorder is present in 10.4% of patients.
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Bruns nystagmus is an unusual type of bilateral nystagmus most commonly occurring in patients with cerebellopontine angle tumours. It is caused by the combination of slow, large amplitude nystagmus (gaze paretic nystagmus) when looking towards the side of the lesion, and rapid, small amplitude nystagmus (vestibular nystagmus) when looking away from the side of the lesion. It occurs in 11% of patients with vestibular schwannoma, and occurs mainly in patients with larger tumours (67% of patients with tumours over 3.5 cm diameter). Bruns nystagmus is also associated with an increased incidence of balance disturbance in patients with vestibular schwannoma.
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A physiotherapy program will improve joint range of motion of the paretic limb using passive range of motion exercises. When increases in activity are tolerated, and stability improvements are made, patients will progress from rolling to side-lying, to standing (with progressions to prone, quadruped, bridging, long-sitting and kneeling for example) and learn to transfer safely (from their bed to a chair or from a wheel chair to a car for example). Assistance and ambulation aids are used as required as the patient begins walking and lessened as function increases. Furthermore, splints and braces can be used to support limbs and joints to prevent or treat complications such as contractures and spasticity.
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Pusher syndrome is a clinical disorder following left or right brain damage in which patients actively push their weight away from the nonhemiparetic side to the hemiparetic side. In contrast to most stroke patients, who typically prefer more weight-bearing on their nonhemiparetic side, this abnormal condition can vary in severity and leads to a loss of postural balance. The lesion involved in this syndrome is thought to be in the posterior thalamus on either side, or multiple areas of the right cerebral hemisphere. With a diagnosis of pusher behaviour, three important variables should be seen, the most obvious of which is spontaneous body posture of a longitudinal tilt of the torso toward the paretic side of the body occurring on a regular basis and not only on occasion.
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Nor was Moniz the only medical practitioner in the 1930s to have contemplated procedures directly targeting the frontal lobes. Although ultimately discounting brain surgery as carrying too much risk, physicians and neurologists such as William Mayo, Thierry de Martel, Richard Brickner, and Leo Davidoff had, before 1935, entertained the proposition. Inspired by Julius Wagner-Jauregg's development of malarial therapy for the treatment of general paresis of the insane, the French physician Maurice Ducosté reported in 1932 that he had injected 5 ml of malarial blood directly into the frontal lobes of over 100 paretic patients through holes drilled into the skull. He claimed that the injected paretics showed signs of "uncontestable mental and physical amelioration" and that the results for psychotic patients undergoing the procedure was also "encouraging".
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Botulinum toxin injection is commonly used for small and moderate degrees of infantile esotropia, acquired adult strabismus, and where it is a consequence of retinal detachment surgery, that is, in cases where there is good potential for binocular vision, so that the corrected alignment can be stabilized by motor fusion. Sixth nerve palsy, paralysis of the lateral rectus, the muscle that rotates the eye outwards, is most frequently caused by an ischemic event, from which there is frequently substantial recovery. But during the acute stage of paresis, the lateral rectus is stretched and grows longer, and its antagonist medial rectus shortens. Sixth nerve palsy is treated by injecting the medial rectus muscle, thereby allowing the lateral rectus, paretic though it be, to stretch and lengthen the medial, while it shortens, so that, when the sixth nerve paresis subsides, alignment is improved.
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