As the vodka dries, its smell will dissipate, taking lingering malodor with it.
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They suggest using a detergent designed to combat malodor, like their own Tide Sport Odor Defense.
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P & G, the parent company of Tide, Gain and Downy, devotes no small amount of time and research dollars to the issue of awful smelling clothes, or malodor, as it's more formally known.
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Here's what they found: 12 of 18 of the key malodor molecules that contributed to the bouquet of that vintage smell were derived from body soils, which is a gentle way of saying your skin, your sweat, your oils. Distressing!
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Ty MatejowskyAssociate Professor, Anthropology, University of Central FloridaDurian, aka "the king of fruits," remains one of the most controversial foods presently consumed in Southeast Asia, and increasingly around the world—not just for its high sulfur content and strong malodor (it's banned from many hotels, airlines, and other public places, and sometimes referred to as the "blue cheese of fruits") but also for the dangers it poses to those who eat, cultivate, or come in close proximity to it.
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Starting at puberty, males have higher levels of androgens than do females and produce comparatively more axillary malodor. As such, it has been proposed that the higher axillary malodor seen in males is due to greater relative stimulation of axillary apocrine sweat glands by androgens.
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An unpleasant odor can also be described as "reeking" or called a "malodor", "stench", or "pong".
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Zola and his gang delved into moral cesspools, and the world grew aweary of the malodor.
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Secretions from sweat glands are initially odorless, but preodoriferous compounds or malodor precursors in the secretions are transformed by skin surface bacteria into volatile odorous compounds that are responsible for body malodor. Water and nutrients secreted by sweat glands also contribute to body odor by creating an ideal environment for supporting the growth of skin surface bacteria.
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It may be asymptomatic, but it can cause rectal pain, rectal bleeding, rectal malodor, incomplete evacuation and obstructed defecation (rectal outlet obstruction).
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Dr. Joseph Tonzetich (deceased) is considered the modern-day pioneer in bad breath research. During the 1960s and 1970s in particular, Tonzetich and colleagues established that volatile sulfur-containing compounds were key identifiable gases in oral malodor. He also provided quantitative support for the hypothesis proposed by G.L.Grapp in the early 1930s that the back of the tongue is the major source of oral malodor. Tonzetich was a professor (1968–1990) in the Faculty of Dentistry, University of British Columbia, and donated more than $300,000 to establish a fellowship endowment fund at the university.
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Androstenol, a 16-androstene pheromone. 16-Androstenes, or androst-16-enes, are a class of endogenous androstane steroids that includes androstadienol, androstadienone, androstenone, and androstenol, which are pheromones. Some of the 16-androstenes, such as androstenone and androstenol, are odorous, and have been confirmed to contribute to human malodor.
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This can be particularly strong when it happens in the axillary region (underarms). In this case, the condition may be referred to an axillary osmidrosis. Trimethylaminuria (TMAU), also known as fish odor syndrome or fish malodor syndrome, is a rare metabolic disorder where trimethylamine is released in the person's sweat, urine, and breath, giving off a strong fishy odor or strong body odor.
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By combining metal salts and polyaniline nanofibers, detection of hydrogen sulfide can be performed. Another study decorated polyaniline nanofibers with gold nanoparticles to detect volatile sulfur compounds in expired human breath.Liu, C.; Hayashi, K.; Toko, K.; Sens. Actuators B, 2012, 161, 504-509() These sensors can potentially be used in various breath analyses and also in disease diagnosis for diseases with malodor biomarker gases.
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Sanguinarine-containing mouthwashes are marketed as anti-plaque and anti-malodor. It is a toxic alkaloid herbal extract, obtained from plants such as Sanguinaria canadensis (Bloodroot), Argemone mexicana (Mexican Prickly Poppy) and others. However, its use is strongly associated with development of leukoplakia (a white patch in the mouth), usually in the buccal sulcus.Leukoplakia , (pdf format) hosted by the American Academy of Oral and Maxillofacial Pathology.
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Hairy tongue largely occurs in the central part of the dorsal tongue, just anterior (in front) of the circumvallate papillae, although sometimes the entire dorsal surface may be involved. Discoloration usually accompanies hairy tongue, and may be yellow, brown or black. Apart from the appearance, the condition is typically asymptomatic, but sometimes people may experience a gagging sensation or a bad taste. There may also be associated oral malodor (intra-oral halitosis).
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All sewage from the new buildings drained downhill and into the Gowanus. Since there was less open ground than before, rainwater now went onto the roofs of the buildings and down into the canal. The building of new sewer connections only compounded the problem by discharging raw sewage from farther away neighborhoods into the canal. Pollutants, storm runoff, and discharge from the sewage system combined to make the canal's malodor so disgusting it was nicknamed "Lavender Lake".
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George Preti (October 7, 1944 – March 3, 2020) was an analytical organic chemist who worked at the Monell Chemical Senses Center in Philadelphia, Pennsylvania. For more than four decades, his research focused on the nature, origin, and functional significance of human odors. Dr. Preti's laboratory has identified characteristic underarm odorants, and his later studies centered upon a bioassay-guided approach to the identification of human pheromones, odors diagnostic of human disease, human malodor identification and suppression and examining the “odor-print” of humans.
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Bismuth subgallate, with a chemical formula C7H5BiO6, is commonly used to treat malodor by deodorizing flatulence and stools. In the United States, it (bismuth subgallate) is the active ingredient in Devrom (internal deodorant), an over-the-counter FDA-approved medicine. Also, it has been used to treat Helicobacter pylori infection and is used in wound therapy. As an internal deodorant, it is commonly used by individuals who have had gastrointestinal stoma surgery, bariatric surgery, fecal incontinence, and irritable bowel syndrome.
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The BANA test (referring to the enzymatic breakdown of [N-benzoyl-dL- arginine-2-napthylamide]) is used to determine the proteolytic activity of certain oral anaerobes that contribute to oral malodor. Some bacteria, e.g. Prophyromona gingivalis, Treponema denticola, and Bacteroides forsythus (Red complex) produce waste products that are quite odiferous, and as a result contribute to bad breath. When a sample of a patient's saliva that contains these bacteria is placed within the BANA testing compound, it causes the breakdown of the N-benzoyl enzyme.
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There are three types of sweat glands: eccrine, apocrine, and apoeccrine. Apocrine glands are primarily responsible for body malodor and, along with apoeccrine glands, are mostly expressed in the axillary (underarm) regions, whereas eccrine glands are distributed throughout virtually all of the rest of the skin in the body, although they are also particularly expressed in the axillary regions, and contribute to malodor to a relatively minor extent. Sebaceous glands, another type of secretory gland, are not sweat glands but instead secrete sebum (an oily substance), and may also contribute to body odor to some degree. The main odorous compounds that contribute to axillary odor include: # Unsaturated or hydroxylated branched fatty acids, with the key ones being (E)-3-methyl-2-hexenoic acid (3M2H) and 3-hydroxy-3-methylhexanoic acid (HMHA) # Sulfanylalkanols, particularly 3-methyl-3-sulfanylhexan-1-ol (3M3SH) # Odoriferous androstane steroids, namely the pheromones androstenone (5α-androst-16-en-3-one) and androstenol (5α-androst-16-en-3α-ol) These malodorous compounds are formed from non-odoriferous precursors that are secreted from apocrine glands and converted by various enzymes expressed in skin surface bacteria.
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Many of the sakacins have been tested for industrial applications and inserted into other lactic acid bacteria. Some have been engineered for production in food environments as well. Many were actually discovered in food contexts, like Greek dry cured sausage (sakacin B). In modern food chemistry, the sakacins have been studied for their use against Listeria in the production of sausages (like Portuguese lingüiça) and cured meat products (such as ham and cold cuts), cheeses, and other lactic acid fermented products. They are also used to repress unwanted bacterial growth that might cause ropiness, sliminess, malodor and other product defects.
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Chlorhexidine is rarely associated with other issues like overgrowth of enterobacteria in persons with leukemia, desquamation and irritation of oral mucosa, salivary gland pain and swelling, and hypersensitivity reactions including anaphylaxis. A randomized clinical trial conducted in Rabat university in Morocco found better results in plaque inhibition when chlorohexidine with alcohol base 0.12% was used, when compared to an alcohol free 0.1% chlorhexidine mouthrinse. Chlorhexidine mouthrinses increase staining score of teeth over a period of time. Hexetidine also has anti-plaque, analgesic, astringent and anti-malodor properties but is considered as an inferior alternative to Chlorhexidine.
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A prank stink bomb and its packaging A stink bomb is a device designed to create an unpleasant smell. They range in effectiveness from simple pranks to military grade malodorants or riot control chemical agents. The Guinness Book of Records lists two smelliest substances: one is "US Government Standard Bathroom Malodor"; a mixture of eight chemicals with a stench resembling human feces, only much stronger, designed to test the efficacy of deodorizers and air fresheners, and the other one, "Who me?", is a mixture of five sulfur- containing chemicals and smells like rotting food and carcasses..
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In addition to having published dozens of peer-reviewed research articles, Dr. Preti held more than a dozen patents related to deodorance, odor mediated control of the menstrual cycle, and the use of odors in disease diagnosis. His unique area of research resulted in hundreds of clinician- directed referrals of patients with an idiopathic body and oral malodor production problems. His efforts in this area revealed a large, undiagnosed population of people suffering from trimethylaminuria, an odor-producing genetic disorder. Preti’s work has frequently been cited by the news media, including the New York Times magazine section, the Philadelphia Inquirer, and ABC’s “Primetime: Medical Mysteries”.
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The specific skin surface bacteria responsible are mainly Staphylococcus and Corynebacterium species. The androstane steroids dehydroepiandrosterone sulfate (DHEA-S) and androsterone sulfate have been detected in an extract of axillary hairs together with high concentrations of cholesterol. Apocrine sweat contains relatively high amounts of androgens, for instance dehydroepiandrosterone (DHEA), androsterone, and testosterone, and the androgen receptor (AR), the biological target of androgens, is strongly expressed in the secretory cells of apocrine glands. In addition, 5α-reductase type I, an enzyme which converts testosterone into the more potent androgen dihydrotestosterone (DHT), has been found to be highly expressed in the apocrine glands of adolescents, and DHT has been found to specifically contribute to malodor as well.
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Trimethylaminuria (TMAU), also known as fish odor syndrome or fish malodor syndrome, is a rare metabolic disorder that causes a defect in the normal production of an enzyme named flavin-containing monooxygenase 3 (FMO3). When FMO3 is not working correctly or if not enough enzyme is produced, the body loses the ability to properly convert trimethylamine (TMA) from precursor compounds in food digestion into trimethylamine oxide (TMAO), through a process called N-oxidation. Trimethylamine then builds up and is released in the person's sweat, urine, and breath, giving off a strong fishy odor or strong body odor. A variant of TMAU (secondary trimethylaminuria or TMAU2) exists where there is no genetic cause, yet excessive TMA is secreted, possibly due to intestinal dysbiosis, altered metabolism, or hormonal causes.
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The excessive amounts of nitrogen from the waste can also contribute to acid rain in the local areas; team of scientists from the US Agricultural Research Service and the US Department of the Environment has examined and noted that within wastewater lagoons in North and South Carolina, there are a host of genes involved in the process of turning ammonia into nitrogen. One case study, conducted by Environmental Health Perspectives, sought to prove that malodor and pollutant concentrations from swine operations are associated with stress, altered mood, and increased blood pressure. For two weeks, adult volunteers living near swine operations in North Carolina sat outside for ten minutes twice a day. They reported levels of hog odor, and recorded their blood pressure.
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The low cost of SDS, its lack of impact on taste, its potential impact on volatile sulfur compounds (VSCs), which contribute to malodorous breath, and its desirable action as a foaming agent have led to the use of SDS in the formulations of toothpastes. A series of small crossover studies (25-34 patients) have supported the efficacy of SLS in the reduction of VSCs, and its related positive impact on breath malodor, although these studies have been generally noted to reflect technical challenges in the control of study design variables. While primary sources from the group of Irma Rantanen at University of Turku, Finland conclude an impact on dry mouth (xerostomia) from SLS-containing pastes, a 2011 Cochrane review of these studies, and of the more general area, concludes that there "is no strong evidence… that any topical therapy is effective for relieving the symptom of dry mouth."See See Rantanen, et al. (2003) J. Contemp. Dent. Pract. 4(2):11–23, , and Rantanen, et al.
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In addition to earwax phenotype, the ABCC11 genotype has been found to be associated with colostrum secretion from the breasts as well as normal axillary odor and osmidrosis (excessive axillary malodor). A functional ABCC11 protein has been found to be essential for the presence of the characteristic strong axillary odor, with the 538G→A SNP leading to a loss of secretion of axillary malodorous precursors and a nearly complete loss of axillary odor in those who are homozygous for the polymorphism. Specifically, the secretion of the amino-acid conjugates 3M2H-Gln, HMHA-Gln, and Cys-Gly-(S) 3M3SH, which are precursors of key axillary malodorous compounds including the unsaturated or hydroxylated branched-chain fatty acids 3M2H and HMHA and the sulfanylalkanol 3M3SH, has been found to be abolished in homozygotic carriers of the SNP, and the odoriferous androstane steroids androstenone and androstenol and their precursors DHEA and DHEA-S have been found to be significantly reduced as well. Patients with axillary osmidrosis (538G/G or 538G/A genotype) were found to have significantly more numerous and relatively large axillary apocrine glands compared to controls with the A/A genotype.
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