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"inappetence" Definitions
  1. loss or lack of appetite
"inappetence" Synonyms

13 Sentences With "inappetence"

How to use inappetence in a sentence? Find typical usage patterns (collocations)/phrases/context for "inappetence" and check conjugation/comparative form for "inappetence". Mastering all the usages of "inappetence" from sentence examples published by news publications.

The symptom also occurs in other animals, such as cats, dogs, cattle, goats, and sheep. In these species, anorexia may be referred to as inappetence. As in humans, loss of appetite can be due to a range of diseases and conditions, as well as environmental and psychological factors. The term is from (, 'without' + , spelled , meaning 'appetite').
Clinical signs may vary, with regurgitation and neurological symptoms being the most prominent in the early and later stages of its progression, respectively. In boa constrictors, the first signs may include off-and-on regurgitation followed by inappetence, and some develop head tremors. Abnormal shedding may occur. Some develop chronic regurgitation and anorexia (lack of appetite or refusal to feed).
Salt toxicosis also affects many breeds of pets or domesticated animals, such as cats, dogs, horses, cows, and birds. Symptoms for pets may include vomiting, diarrhea, inappetence, lethargy, walking drunk, abnormal fluid accumulation within the body, excessive thirst or urination, potential injury to the kidneys, tremors, seizures, coma, and eventually could lead to death without care. Salt toxicosis may be caused by the ingestion of salt dough.
They are said to cause vomiting, inappetence, lethargy, kidney failure, and even death. Cats are the only species known to be affected. The National Animal Poison Control Center says that certain types of lilies can cause kidney failure in cats that have ingested any part of the lily. The Society of American Florists, a floral industry umbrella organization, recommends keeping lilies out of the reach of cats.
Ahmed S.A., Amin A.E., Adam S.E., Hapke H.J. By toxic effects of the dried leaves and stem of Capparis tomentosa on Nubian goats. Dtsch Tierarztl Wochenschr. 1993 May;100(5):192-4. Signs of Capparis poisoning in the sheep and calves were; weakness of the hind limbs, staggering, swaying, flexion of the fetlock and phalangeal joints, pain in the sacral region, inappetence and recumbency.
The most common form of the disease is the head and eye form. Typical symptoms of this form include fever, depression, discharge from the eyes and nose, lesions of the buccal cavity and muzzle, swelling of the lymph nodes, opacity of the corneas leading to blindness, inappetence and diarrhea. Some animals have neurologic signs, such as ataxia, nystagmus, and head pressing. Animals that become infected with the virus can become extremely sensitive to touch, especially around the head.
In dogs, adverse effects may include lethargy, decreased appetite, vomiting, diarrhea, blood in feces, and flatulence. In cats, adverse reactions may include vomiting, diarrhea, decreased appetite, lethargy, odd hyperactive behavior, and inappropriate urination. Mildly increased serum alanine transaminase (ALT) and gamma- glutamyltransferase may also occur. Other reported events in dogs and cats include death, tremors/ataxia, seizures, anaphylaxis, acute pulmonary edema, facial edema, injection site reactions (alopecia, scabs, necrosis, and erythema), hemolytic anemia, salivation, pruritus, lethargy, vomiting, diarrhea, and inappetence.
Some major clinical signs of M. gallisepticum in chickens include those of respiratory distress such as coughing, sneezing, slight to marked rales, and difficulty breathing."Mycoplasma gallisepticum Infection in Poultry" in The Merck Veterinary Manual for Veterinary Professionals (2013). Swollen eyelids, ocular discharge, and loss of sight are signs and symptoms that are very important for this disease as well. Poor productivity, leg problems, nasal discharge, stunting, inappetence, slow growth, reduced hatchability, reduced chick viability, and abnormal feathers are also some relevant clinical signs of the disease.
Signs and symptoms of PHF include acute-onset fever, depression (sometimes profound), inappetence, mild colic-like symptoms, decreased manure production, profuse watery non-fetid diarrhea endotoxemia, edema due to protein imbalances, abortion by pregnant mares, and acute laminitis (20 to 40 percent of cases). Infected horses founder usually within three days of the initial symptoms, thought to be secondary to endotoxemia. Death may occur and is usually due to severe laminitis leading to founder. Horses may not always display any other symptoms beyond a fever.
Roe deer fawn, two to three weeks old The polygamous roe deer males clash over territory in early summer and mate in early autumn. During courtship, when the males chase the females, they often flatten the underbrush, leaving behind areas of the forest in the shape of a figure eight called 'roe rings'. Males may also use their antlers to shovel around fallen foliage and soil as a way of attracting a mate. Roebucks enter rutting inappetence during the July and August breeding season.
Most infected cats have been healthy before a very sudden onset of severe disease. The course of clinical disease is often swift with clinical signs of lethargy and inappetence within 5 to 20 days after the tick bite. Cats develop a high fever, but the temperature may become low before death. Other clinical findings can be: dehydration, icterus (jaundice), enlarged liver and spleen, lymphadenopathy, pale mucus membranes, respiratory distress, tachycardia or bradycardia, and tick infestation (although ticks are not often found on infected cats since cats typically groom ticks off their fur).
There was a decrease in the level of total protein and calcium and an increase of glutamic oxaloacetic transaminase (GOT), ammonia, sodium and potassium in serum. The main pathological changes were vacuolation of the neurons and axons in the spinal cord, with necrosis of the centrilobular hepatocytes and renal convoluted tubules and glomeruli. In Capparis-fed goats, anaemia developed and the results of kidney and liver function tests were correlated with clinical abnormalities and pathologic changes. The prominent features of toxicity were inappetence, locomotor disturbances, paresis especially of the hind limbs and recumbency.
The name equine encephalosis is misleading as the disease is not primarily a neurological disorder. Although the majority of infections result only in mild clinical signs, in more severe cases clinical signs include a short period (typically two to five days) of fluctuating fever, accompanied by varying degrees of inappetence. Elevated heart and respiratory rates are also common, and occasionally as a result of nasal congestion, a red-brown discolouration of the mucous membranes may be observed. Although rare, more severe clinical signs may occur including facial swelling (lips and eyelids), respiratory distress, and petechial haemorrhages of the conjunctivae.

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