The CDC describes the effects of eating toxic blowfish thusly: First stage: Numbness and sensation of prickling and tingling (paresthesia) of the lips and tongue, followed by facial and extremity paresthesias and numbness, headache, sensations of lightness or floating, profuse sweating (diaphoresis), dizziness, salivation (ptyalism), nausea, vomiting (emesis), diarrhea, abdominal (epigastric) pain, difficulty moving (motor dysfunction), weakness (malaise), and speech difficulties.
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Most infections can cause some degree of diaphoresis and it is a very common symptom in some serious infections such as malaria and tuberculosis. In addition, pneumothorax can cause diaphoresis with splinting of the chest wall. Neuroleptic malignant syndrome and other malignant diseases (e.g. leukemias) can also cause diaphoresis.
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Diaphoresis may be associated with some abnormal conditions, such as hyperthyroidism and shock. If it is accompanied by unexplained weight loss or fever or by palpitations, shortness of breath, or chest discomfort, it suggests serious illness. Diaphoresis is also seen in an acute myocardial infarction (heart attack), from the increased firing of the sympathetic nervous system, and is frequent in serotonin syndrome. Diaphoresis can also be caused by many types of infections, often accompanied by fever and/or chills.
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Diabetics relying on insulin shots or oral medications may have low blood sugar (hypoglycemia), which can also cause diaphoresis. Drugs (including caffeine, morphine, alcohol, antidepressants and certain antipsychotics) may be causes, as well as withdrawal from alcohol, benzodiazepines, nonbenzodiazepines or narcotic painkiller dependencies. Sympathetic nervous system stimulants such as cocaine and amphetamines have also been associated with diaphoresis. Diaphoresis due to ectopic catecholamine is a classic symptom of a pheochromocytoma, a rare tumor of the adrenal gland.
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If not treated, there is a 30% risk of bleeding. Bleeding may lead to hypotension, tachycardia, and sweating (diaphoresis).
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A man in a sweat-drenched shirt, presumably after some physical exertion. Diaphoresis is a non-specific symptom or sign, which means that it has many possible causes. Some causes of diaphoresis include physical exertion, menopause, fever, ingestion of toxins or irritants, and high environmental temperature. Strong emotions (anger, fear, anxiety) and recall of past trauma can also trigger sweating.
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Both the words diaphoresis and hidrosis can mean either perspiration (in which sense they are synonymous with sweating) or excessive perspiration, in which case they refer to a specific, narrowly defined, clinical disorder.
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Key signs of excited delirium are aggression, altered mental status, and diaphoresis/hyperthermia. Other conditions which can resemble excited delirium are mania, neuroleptic malignant syndrome, hypoglycemia, thyroid storm, and catatonia of the malignant or excited type.
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The symptoms of a cholinergic toxidrome include bronchorrhea, confusion, defecation, diaphoresis, diarrhea, emesis, lacrimation, miosis, muscle fasciculations, salivation, seizures, urination, and weakness. Complications include bradycardia, hypothermia, and tachypnea. Substances that may cause this toxidrome include carbamates, mushrooms, and organophosphates.
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Metastatic insulinomas are commonly preceded by hypoglycemic symptoms and the Whipple triad. Majority of the hypoglycemic symptoms will manifest as neuroglycopenic symptoms and/or autonomic symptoms. Common neuroglycopenic symptoms include: generalized confusion, significant behavioral changes, coma, and seizure. Common autonomic symptoms include: diaphoresis, weakness, palpitations, and hunger.
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Side effects associated with ospemifene include vaginal discharge, hot flashes, and diaphoresis. More serious adverse effects are similar to those of estrogens and estrogen receptor modulators. These include, but are not limited to, thromboembolism, allergic reactions, fatigue, and headache, and others could occur. There are other additional adverse effects.
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The first published case of paroxysmal sympathetic hyperactivity was Wilder Penfield's case report of a 41-year-old woman, JH, published in 1929. She had a third ventricle cholesteatoma. She displayed increased respiration, increased heart rate, diaphoresis, and increased blood pressure. She also displayed minor symptoms: pupillary dilation, hiccups, and lacrimation.
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The FDA has classified the side effects into groups based on dosages levels at q4h. For the high dosage group (90 mg) less than 1% of the group experienced adverse conditions including itching, gastrointestinal hemorrhage, thrombocytopenia, neurological deterioration, vomiting, diaphoresis, congestive heart failure, hyponatremia, decreasing platelet count, disseminated intravascular coagulation, deep vein thrombosis.
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Huoxiang Zhengqi Shui () is a liquid herbal formula used in Traditional Chinese medicine to "induce diaphoresis and clear away summer-heat, to resolve damp and regulate the function of the spleen and stomach".State Pharmacopoeia Commission of the PRC (2005). "Pharmacopoeia of The People's Republic of China (Volume I)". Chemical Industry Press. .
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The usual treatment of a standardised Adie syndrome is to prescribe reading glasses to correct for impairment of the eye(s). Pilocarpine drops may be administered as a treatment as well as a diagnostic measure. Thoracic sympathectomy is the definitive treatment of diaphoresis, if the condition is not treatable by drug therapy.
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The symptoms of a sympathomimetic toxidrome include anxiety, delusions, diaphoresis, hyperreflexia, mydriasis, paranoia, piloerection, and seizures. Complications include hypertension, and tachycardia. Substances that may cause this toxidrome include cocaine, amphetamine, and compounds based upon amphetamine's structure such as ephedrine (Ma Huang), methamphetamine, phenylpropanolamine (PPA), and pseudoephedrine. The bronchodilator salbutamol may also cause this toxidrome.
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The following side effects are possible: anxiety, panic attacks, dysphoria, depression, agitation, irritability, suicidal ideation, fatigue, orthostatic hypotension, nausea, vomiting, diaphoresis, generalised pain, and drug cravings. For some individuals, these withdrawal symptoms are short-lived and make a full recovery, for others a protracted withdrawal syndrome may occur with withdrawal symptoms persisting for months or years.
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A chest X-ray might depict pleural effusion, pulmonary infiltration, or pericardial effusion. During medical doctor examination, a pericardial friction rub can be auscultated indicating pericarditis. Auscultation of the lungs can show crackles indicating pulmonary infiltration, and there can be retrosternal/pleuritic chest pain worse on inspiration (breathing in). Patient can also depict sweating (diaphoresis) and agitation or anxiety.
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Cyclopaedia: or, An Universal Dictionary of Arts and Sciences, Vol. 1, London 1728: 318. It also says that the root and the recipe Lapis Contrayerva (see below) are of great efficacy in smallpox, measles, fevers and in “all Cases where either a Diaphoresis or Perspiration is required.”Chambers, Ephraim. Cyclopaedia: or, An Universal Dictionary of Arts and Sciences, Vol. 1, London 1728: 319.
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Vagotonia is the state of the autonomic nervous system in which the equilibrium between the sympathetic and parasympathetic nervous system is biased towards the parasympathetic, the opposite phenomenon being sympatheticotonia. There is an associated clinical syndrome with low blood pressure (hypotension), low heart rate (bradycardia), miosis, often cold hands and feet, a cold and clammy diaphoresis, severe fatigue, and sometimes vasovagal syncope.
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In the review of systems, obtain a complete description of any pain in the upper abdomen or epigastric area. Symptoms that may be important in relation to pancreatic disorders are pruritus, abdominal pain, dyspnea, nausea, and vomiting. The functional assessment includes data about the patient’s dietary habits and use of alcohol. Note any restlessness, flushing, or diaphoresis during the examination.
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Despite the venom lacking the atraxotoxin or atraxin of A. robustus, the symptoms are very similar to those from a Sydney funnel-web spider bite. Common symptoms include diaphoresis (profuse sweating), hypertension (elevated blood pressure), sinus tachycardia (elevated heart rate), nausea, vomiting and local pain at the bite site. Pulmonary oedema (fluid build-up in the lungs) often comes on early.
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Relapsing fever is easily treated with a one- to two-week-course of antibiotics, and most people improve within 24 hours. Complications and death due to relapsing fever are rare. Tetracycline-class antibiotics are most effective. These can, however, induce a Jarisch–Herxheimer reaction in over half those treated, producing anxiety, diaphoresis, fever, tachycardia and tachypnea with an initial pressor response followed rapidly by hypotension.
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Yinqiao Jiedu Wan (, Pinyin: yín qiáo jiě dú wán) is a brown pill used in Traditional Chinese medicine to "induce diaphoresis, remove heat and counteract toxicity". It is aromatic, and it tastes bitter, pungent and slightly sweet. It is used where there is "upper respiratory infection with fever, headache, cough, dryness of the mouth and sore throat".State Pharmacopoeia Commission of the PRC (2005).
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The adverse effects of pethidine administration are primarily those of the opioids as a class: nausea, vomiting, dizziness, diaphoresis, urinary retention, and constipation. Due to moderate stimulant effects mediated by dopamine and norepinephrine, sedation is less likely compared to other opioids. Unlike other opioids, it does not cause miosis because of its anticholinergic properties. Overdose can cause muscle flaccidity, respiratory depression, obtundation, cold and clammy skin, hypotension, and coma.
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Non-selective beta-blockers are the most effective in reducing the frequency and severity of PSH episodes. They help decrease the effect of circulating catecholamines and lower metabolic rates, which are high in patients during PSH episodes. Beta-blockers also help in reducing fever, diaphoresis, and in some cases dystonia. Propanolol is a common beta-blocker administered due to the fact that it penetrates the blood-brain barrier relatively well.
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Brain activities during a typical episode show theta and alpha activity when monitored with an EEG. Episodes can include tachycardia. Night terrors are also associated with intense autonomic discharge of tachypnea, flushing, diaphoresis, and mydriasis—that is, unconscious or involuntary rapid breathing, reddening of the skin, profuse sweating, and dilation of the pupils. Abrupt but calmer arousal from NREM sleep, short of a full night-terror episode, is also common.
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Paroxysmal sympathetic hyperactivity (PSH) is a syndrome that causes episodes of increased activity of the sympathetic nervous system. Hyperactivity of the sympathetic nervous system can manifest as increased heart rate, increased respiration, increased blood pressure, diaphoresis, and hyperthermia. Previously, this syndrome has been identified as general dysautonomia but now is considered a specific form of it. It has also been referred to as paroxysmal sympathetic instability with dystonia, or PAID, and sympathetic storm.
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After long-term use of dopamine agonists, a withdrawal syndrome may occur during dose reduction or discontinuation with the following possible side effects: anxiety, panic attacks, dysphoria, depression, agitation, irritability, suicidal ideation, fatigue, orthostatic hypotension, nausea, vomiting, diaphoresis, generalized pain, and drug cravings. For some individuals, these withdrawal symptoms are short-lived and they make a full recovery, for others a protracted withdrawal syndrome may occur with withdrawal symptoms persisting for months or years.
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The classic sign of pericarditis is a friction rub heard with a stethoscope on the cardiovascular examination, usually on the lower left sternal border. Other physical signs include a person in distress, positional chest pain, diaphoresis (excessive sweating); possibility of heart failure in form of pericardial tamponade causing pulsus paradoxus, and the Beck's triad of low blood pressure (due to decreased cardiac output), distant (muffled) heart sounds, and distension of the jugular vein (JVD).
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After long-term use of dopamine agonists, a withdrawal syndrome may occur during dose reduction or discontinuation with the following possible side effects: anxiety, panic attacks, dysphoria, depression, agitation, irritability, suicidal ideation, fatigue, orthostatic hypotension, nausea, vomiting, diaphoresis, generalized pain, and drug cravings. For some individuals, these withdrawal symptoms are short-lived and make a full recovery, for others a protracted withdrawal syndrome may occur with withdrawal symptoms persisting for months or years.
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People who might orally ingest acute amounts would experience emesis, diaphoresis, drowsiness and disorientation. This would need to be intentional since a large amount would need to be ingested to experience a toxic reaction. In dogs the LD50 is 450 mg/kg of body weight (i.e., in any sample of medium-sized dogs weighing , half of them would be killed after consuming 5,850 mg of imidacloprid, or about th of an ounce) .
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The abuse of exogenous insulin carries with it an attendant risk of hypoglycemic coma and death when the amount used is in excess of that required to handle ingested carbohydrate. Acute risks include brain damage, paralysis, and death. Symptoms may include dizziness, weakness, trembling, palpitations, seizures, confusion, headache, drowsiness, coma, diaphoresis and nausea. All persons suffering from overdoses should be referred for medical assessment and treatment, which may last for hours or days.
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Acetylcholinesterase inhibitors (e.g. some insecticides) also cause contraction of sweat gland smooth muscle leading to diaphoresis. Mercury is well known for its use as a diaphoretic, and was widely used in the 19th and early 20th century by physicians to "purge" the body of an illness. However, due to the high toxicity of mercury, secondary symptoms would manifest, which were erroneously attributed to the former disease that was being treated with mercurials.
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The effects of organophosphate poisoning on muscarinic receptors are recalled using the mnemonic SLUDGEM (salivation, lacrimation, urination, defecation, gastrointestinal motility, emesis, miosis) An additional mnemonic is MUDDLES: miosis, urination, diarrhea, diaphoresis, lacrimation, excitation, and salivation. The onset and severity of symptoms, whether acute or chronic, depends upon the specific chemical, the route of exposure (skin, lungs, or GI tract), the dose, and the individuals ability to degrade the compound, which the PON1 enzyme level will affect.
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Orthostatic syncope may occur suddenly with no warning or may be preceded by symptoms. Associated symptoms are usually because of cerebral hypoperfusion occurring in the upright position and include dizziness, feeling faint or nauseated, diaphoresis, a sense of warmth or blurred vision. Other general symptoms regardless of the position include a feeling of generalized weakness, headache, fatigue, cognitive slowing and shortness of breath. Symptoms may be sudden or gradual, getting progressively worse until the patient loses consciousness.
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Generally, labile hypertension does not present any physical symptoms during fluctuations of blood pressure. Elevation and decrease of blood pressure reading usually occurs without intervention. However, if the normal resting pressure remains abnormally excessive, common signs and symptoms that are present include: Headache, Heart palpitations, Flushing, Tinnitus (ringing or buzzing noise in one or both ears), Weakness of body, Dizziness and diaphoresis. Some may describe the episodes as abrupt in onset such that they were not evoked by stress.
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Use over the recommended dose of about 120 μg can cause muscle tremors, headache, dizziness, and gastric irritation. Persons self-administering the drug for weight loss or to improve athletic performance have experienced nausea, vomiting, diaphoresis, palpitations, tachycardia, and myocardial infarction. Use of the drug may be confirmed by detecting its presence in semen or urine.R. Baselt, Disposition of Toxic Drugs and Chemicals in Man, 8th edition, Biomedical Publications, Foster City, CA, 2008, pp. 325–326.
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In oral rehydration therapy, electrolyte drinks containing sodium and potassium salts replenish the body's water and electrolyte concentrations after dehydration caused by exercise, excessive alcohol consumption, diaphoresis (heavy sweating), diarrhea, vomiting, intoxication or starvation. Athletes exercising in extreme conditions (for three or more hours continuously, e.g. a marathon or triathlon) who do not consume electrolytes risk dehydration (or hyponatremia). A home-made electrolyte drink can be made by using water, sugar and salt in precise proportions.
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Chronic use of alcohol used to induce sleep can lead to insomnia: frequent moving between sleep stages occurs, with awakenings due to headaches and diaphoresis. Stopping chronic alcohol abuse can also lead to profound disturbances of sleep with vivid dreams. Chronic alcohol abuse is associated with NREM stage 3 and 4 sleep as well as suppression of REM sleep and REM sleep fragmentation. During withdrawal REM sleep is typically exaggerated as part of a rebound effect.
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All of these processes result in patients possessing systemic symptoms (chills, fever, diaphoresis), bacteremia, and lymphatic enlargement. A major role in B. quintana infection is its lipopolysaccharide covering which is an antagonist of the toll-like receptor 4. The reason this infection might persist is because this organism also results in monocytes overproducing interleukin-10 (IL-10), thus weakening the immune response. B. quintana also induces lesions seen in bacillary angiomatosis that protrude into vascular lumina, often occluding blood flow.
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The most common presenting symptom is chest pain that is often described as tightness, pressure or squeezing. The onset of symptoms is usually gradual, over several minutes and tends to be located in the central chest (overlying the sternum) although it can be experienced in the left chest, right chest, and even abdominal area. Associated symptoms, which are mostly autonomic in nature, include diaphoresis, nausea, vomiting, palpitations, and anxiety (which is often described as a sense of impending doom).Mallinson, T (2010).
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Despite the venom lacking the atraxotoxin or atraxin of A. robustus, the symptoms are very similar to those from a Sydney funnel-web spider bite. Features of envenomation include diaphoresis, local pain at the bite site, pulmonary oedema, hypertension, nausea and vomiting. Females are suspected of being more venomous than males, possibly because they inject larger amounts of venom. Versutoxin, a neurotoxin very similar to robustoxin produced by the Sydney funnel-web spider, induces an autonomic storm in the victim.
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Human expenditure of energy results in the generation of heat. The body heat generated by normal activities, and particularly by exercise, triggers homeostatic regulatory mechanisms with the goal of maintaining body core temperature within its relatively narrow, safe physiologic range by means of vasoregulation and diaphoresis. The weightlessness environment of spaceflight may impair heat dissipation by reducing evaporative and conductive heat exchange. Microgravity and spaceflight may perturb the body's thermoregulatory mechanisms by altering the work efficiency, metabolic rate, or circadian rhythms of heat production.
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Applying pressure bandages and immobilising the patient can significantly delay the onset of symptoms and remains a critical part of the management of an Australian funnel-web spider bite. Despite the venom lacking the δ-atracotoxin or atraxin of A. robustus, the symptoms are very similar to those from a Sydney funnel- web spider bite. Common symptoms include diaphoresis, hypertension, sinus tachycardia, muscle spasm or fasciculation, nausea and vomiting, altered consciousness and local pain at the bite site. Pulmonary oedema occurs frequently and comes on early.
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A fainting response pattern is not seen in all individuals with BII phobia, but is found in a majority. Up to 80% of those with BII phobia report either syncope or pre-syncope as a symptom when exposed to a trigger. Other symptoms that may evolve when exposed to phobic triggers include extreme chest discomfort, tunnel vision, becoming pale, shock, vertigo, diaphoresis (profuse sweating), nausea, and in very rare cases asystole (cardiac arrest) and death. Increase in stress hormone release (particularly of cortisol and corticotrophin) is typical.
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Opioid analgesics may be necessary to relieve pain. Antivenom has been historically given for adults suffering severe local pain or systemic symptoms consistent with latrodectism, which include pain and swelling spreading proximally from site, distressing local or systemic pain, chest pain, abdominal pain, or excessive sweating (diaphoresis). A significant proportion of bites will not result in envenomation or any symptoms developing; around 2–20% of bite victims have been treated with antivenom. In an Australian study of 750 emergency hospital admissions for spider bites where the spider was definitively identified, 56 were from redbacks.
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When intraperitoneally administered to mice, dinogunellins have a mean lethal dose (LD50) of 25 mg/kg. Dinogunellins are also orally toxic to mice and guinea pigs and has also deleterious effects on humans. A few hours after ingestion, humans develop abrupt onset diaphoresis, chills, abdominal pain and cramping, with nausea and vomiting followed by voluminous, non-bloody diarrhea. Analysis made on the cabezon toxin showed that its effects start 12 hours after administration and is characterized by several signs such as diarrhea, hirsute hair, nasal discharge, and death.
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Careful medical history and physical examination is essential in separating dangerous from trivial causes of disease, and the management of chest pain may be done on specialized units (termed medical assessment units) to concentrate the investigations. Occasionally, invisible medical signs will direct the diagnosis towards particular causes, such as Levine's sign in cardiac ischemia. However, in the case of acute coronary syndrome, a third heart sound, diaphoresis, and hypotension are the most strongly associated physical exam findings. However these signs are limited in their prognostic and diagnostic value.
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The cardinal symptom of critically decreased blood flow to the heart is chest pain, experienced as tightness around or over the chest and (often, but not always) radiating to the left arm and the left angle of the jaw. This may be associated with diaphoresis (sweating), nausea and vomiting, as well as shortness of breath. In many cases, the sensation is "atypical", with pain experienced in different ways or even being completely absent (which is more likely in female patients and those with diabetes). Some may report palpitations, anxiety or a sense of impending doom (angor animi) and a feeling of being acutely ill.
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Both SNRIs have the potential for discontinuation syndrome after abrupt cessation, which can precipitate symptoms including motor disturbances and anxiety and may require tapering. Like other serotonergic agents, SNRIs have the potential to cause serotonin syndrome, a potentially fatal systemic response to serotonergic excess that causes symptoms including agitation, restlessness, confusion, tachycardia, hypertension, mydriasis, ataxia, myoclonus, muscle rigidity, diaphoresis, diarrhea, headache, shivering, goose bumps, high fever, seizures, arrhythmia and unconsciousness. SNRIs like SSRIs carry a black box warning for suicidal ideation, but it is generally considered that the risk of suicide in untreated depression is far higher than the risk of suicide when depression is properly treated.
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It is classified as a snake of medical importance by the World Health Organization. Dangerous snake poster published by Museum Victoria in 1877 Clinically, the venom of the eastern brown snake causes venom-induced consumption coagulopathy; a third of cases develop serious systemic envenoming including hypotension and collapse, thrombotic microangiopathy, severe haemorrhage, and cardiac arrest. Other common systemic symptoms include nausea and vomiting, diaphoresis (sweating), and abdominal pain. Acute kidney injury and seizures can also occur. Onset of symptoms can be rapid, with a headache developing in 15 minutes and clotting abnormalities within 30 minutes; collapse has been recorded as occurring as little as two minutes after being bitten.
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Prolonged exposure to a very high level of nitrogen dioxide in micro meter-size range, may have an inflammatory effect that principally targets the respiratory tracts leading to chronic nitrogen dioxide poisoning which can occur within days or weeks after the threshold limit value is excessively exceeded. This condition causes fever, rapid breathing coupled with rapid heart rate, labored breathing and severe shortness of breath. Other effects include diaphoresis, chest pain, and persistent dry cough, all of which may result in weight loss, anorexia and may also lead to right-side heart enlargement and heart disease in advanced cases. Prolonged exposure to relatively low levels of nitrogen (II) oxide may cause persistent headaches and nausea.
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Dermal exposure resulted in nausea, dizziness, vomiting, headache, or tachycardia. Nicotine poisoning tends to produce symptoms that follow a biphasic pattern. The initial symptoms are mainly due to stimulatory effects and include nausea and vomiting, excessive salivation, abdominal pain, pallor, sweating, hypertension, tachycardia, ataxia, tremor, headache, dizziness, muscle fasciculations, and seizures. After the initial stimulatory phase, a later period of depressor effects can occur and may include symptoms of hypotension and bradycardia, central nervous system depression, coma, muscular weakness and/or paralysis, with difficulty breathing or respiratory failure. From September 1, 2010 to December 31, 2014, there were at least 21,106 traditional cigarette calls to US poison control centers. During the same period, the ten most frequent adverse effects to traditional cigarettes reported to US poison control centers were vomiting (80.0%), nausea (9.2%), drowsiness (7.8%), cough (7.2%), agitation (6.6%), pallor (3.0%), tachycardia (2.5%), diaphoresis (1.5%), dizziness (1.5%), and diarrhea (1.4%).
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A RSBI score of less than 65 indicating a relatively low respiratory rate compared to tidal volume is generally considered as an indication of weaning readiness. A patient with a rapid shallow breathing index (RSBI) of less than 105 has an approximately 80% chance of being successfully extubated, whereas an RSBI of greater than 105 virtually guarantees weaning failure. Other criteria that have been suggested for a successful weaning trial include (1) the ability to tolerate a Spontaneous breathing trial for 30 minutes (in most patients, SBT failure will occur within approximately 20 minutes), (2) maintain a respiration rate of less than 35/min, and (3) keep an oxygen saturation of 90% without arrhythmias; sudden increases in heart rate and blood pressure; or development of respiratory distress, diaphoresis, or anxiety. Once the SBT is tolerated, the ability to clear secretions, a decreasing secretion burden, and a patent upper airway are other criteria that should be met to increase extubation success.
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Knowing a person's risk factors can be extremely useful in ruling in or ruling out serious causes of chest pain. For example, heart attack and thoracic aortic dissection are very rare in healthy individuals under 30 years of age, but significantly more common in individuals with significant risk factors, such as older age, smoking, hypertension, diabetes, history of coronary artery disease or stroke, positive family history (premature atherosclerosis, cholesterol disorders, heart attack at early age), and other risk factors. Chest pain that radiates to one or both shoulders or arms, chest pain that occurs with physical activity, chest pain associated with nausea or vomiting, chest pain accompanied by diaphoresis or sweating, or chest pain described as "pressure," has a higher likelihood of being related to acute coronary syndrome, or inadequate supply of blood to the heart muscle, but even without these symptoms chest pain may be a sign of acute coronary syndrome. Other clues in the history can help lower the suspicion for myocardial infarction.
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